AminoAct™ consists of a propriety set of small protein fragments (peptides) that can be readily absorbed by the body .  These bioactive peptides have been shown to have the following functions:

• AminoAct™ increases the body's overall antioxidant production within one week through the Superoxide Dismutase (SOD) enzyme. SOD is the first line of defense by the body to combat free radicals, thus minimizing cellular damages.
• AminoAct™  stimulates the production of a small hormone in the stomach called Ghrelin.  Ghrelin is known to increase appetite.  
• AminoAct™ reduces inflammation by lowering TNF-alpha, a pro-inflammatory factor in the blood.

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Antioxidants Alleviates Chemotherapy Side Effects

As a person processes food for energy, the body produces free radicals.  These free radicals damage DNA, proteins and lipids, and are believed to contribute to aging, cancer and many other diseases. Cancer patients are reported to have higher levels of oxidative stress^1),2) and some tumor tissues may contain or secrete free radicals³⁾.  These free radicals cause cells to breakdown and promote mutation. 

Furthermore, research has shown that chemotherapy causes the body to have elevated levels of free radicals, and after chemo treatment, the body is depleted of antioxidants.  As a result, vital organs, such as lungs, heart, kidney and liver are often overburdened and injured after chemotherapy treatment. This toxic side effect slows down the health recovery^4),5),6).

A recent clinical review⁷⁾ of 19 clinical studies indicated that antioxidant supplement resulted in either increased survival times, increased treatment responses, or both.  Compared to the control group, patients taking antioxidants also had less side effects from toxicity.

REFERENCES

1. Advanced but not localized prostate cancer is associated with increased oxidative stress.   Journal of Urology.  2007 Oct; 178:1238-43.


2. Oxidative stress and wasting in cancer.  Curr Opin Clin Nutr Metab Care. 2007, 10:449-56.


3. Reactive oxygen species regulate angiogenesis and tumor growth through vascular endothelial growth factor.  Cancer Research.. 2007 Nov 15;67(22):10823-30.


4. Collateral damage in cancer chemotherapy: oxidative stress in non-targeted tissue. Molecular Interventions 2007 Jun; 7:147-156.


5. Oxidative stress and inflammation contribute to lung toxicity after a common breast cancer chemotherapy regimen.  American Journal of Physiology - Lung Cellular and Mollecular Physiology. 2002 283: L336-L345.


6.  Chemotherapy-Associated Oxidative stress: Impact on Chemotherapeutic Effectiveness.  Integrative Cancer Therapies. vol. 3, No. 4, 294-300.


7. Impact of antioxidant supplementation on chemotherapeutic efficacy: a systematic review of the evidence from randomized controlled trials.       Cancer Treat Rev. 2007 Aug: 407-18.